Communications
29 September 2021
Vol. 2 No. 1 (2021)
https://doi.org/10.4081/bse.157
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Superoxide dismutase 1 (SOD1) and cadmium: a three models approach to the comprehension of its neurotoxic effects

Authors

Federica Bovio
Department of Biotechnology and Biosciences, University of Milano-Bicocca, Italy, Milan, Italy.
Barbara Sciandrone
Department of Biotechnology and Biosciences, University of Milano- Bicocca, Italy, Milan, Italy.
Chiara Urani
Department of Earth and Environmental Sciences, University of Milan Bicocca, Milan, Italy; Integrated Models for Prevention and Protection in Environmental and Occupational Health, (MISTRAL), Interuniversity Research Center, Italy.
Paola Fusi
paola.fusi@unimib.it
Department of Biotechnology and Biosciences, University of Milano- Bicocca, Italy, Milan; Integrated Models for Prevention and Protection in Environmental and Occupational Health, (MISTRAL), Interuniversity Research Center, Italy.
Matilde Forcella
Department of Biotechnology and Biosciences, University of Milano- Bicocca, Italy, Milan, Italy.
Maria Elena Regonesi
Department of Biotechnology and Biosciences, University of Milano- Bicocca, Italy, Milan; Milan Center of Neuroscience (NeuroMI), Milan, Italy.

In three different biological models, the recombinant protein expressed in E. coli, the neuronal cells SH-SY5Y and the nematode Caenorhabditis elegans, cadmium inhibits SOD1 activity without affecting its expression level.

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Superoxide dismutase 1 (SOD1) and cadmium: a three models approach to the comprehension of its neurotoxic effects. (2021). Biomedical Science and Engineering, 2(1). https://doi.org/10.4081/bse.157

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